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Monday, June 18, 2018

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Manhunt for Boy with Lymphoma Cancer. Why?  (continued)

Manhunt for Boy with Lymphoma Cancer. Why? For more than a century the disease was not considered a cancer, but was widely regarded as a bacterial and infectious disease, possibly related to the acid-fast bacteria that cause tuberculosis.

Although cancer experts no longer consider bacteria as a cause of HD, various researchers continue to implicate bacteria (not viruses) in this form of cancer.

Not surprisingly, HD and tuberculosis (TB) can co-exist in the same patient [1]; and there is increasing recognition of a close association between HD and so-called "non-Hodgkin's lymphoma"[2]. TB-like bacteria have also been described in "non-Hodgkin's lymphoma," by Busni [3,4], Aplas [5,6], and Cantwell [7,8].

AIDS patients have an increased incidence of both Hodgkin's and non-Hodgkin's lymphoma, as well as a high incidence of Kaposi's sarcoma [9].

Pleomorphic TB-like bacteria have been observed and reported in AIDS-related lymphoma and Kaposi's sarcoma by Cantwell et al. [10-13].

Does chemotherapy and radiation cure Hodgkin's?

Before chemotherapy and radiation treatments were designed for HD, the disease was uniformly fatal. Now the 5-year survival rate for the average patient is about 80%.

However, the patient may play a heavy price healthwise for this standard therapy. Patients who survive radiation treatment for HD can develop a second related cancer.

According to Aisenberg, deaths from second malignancies are the most important cause of death other than HD itself [14].

Women with HD who are radiated have up to a 40 percent greater risk for breast cancer [15]. HD patients are also at increased risk for acute leukemia and non-Hodgkin's lymphoma. University of Texas Southwestern Medical Center researchers have found that patients surviving childhood Hodgkin's disease suffer strokes later in life at rates about four times that of the general population. They suspect the radiation used in treating this cancer as a cause [16].

Because cancer experts claim the cause of all these different cancers is unknown, it is assumed there is no etiologic connection between them.

However, cancer microbe research suggests that
pleomorphic bacteria (and virus-like forms of bacteria) are implicated in many forms of cancer.

Bacteria as a cause of Hodgkin's disease

The microbiology of HD is intimately connected with the microbiology of cancer. The reason for this is that similar-appearing bacteria are found in the tissue in various forms of cancer. In the last half century the leading proponents of the bacteriology of cancer include Virginia Wuerthele-Caspe Livingston, M.D. [17], microbiologist Eleanor Alexander-Jackson [18], cell cytologist Irene Diller [19,20], and noted biochemist Florence Seibert [21,22].

Their work is documented in Livingston's "Cancer: A New Breakthrough" [23] and "The Conquest of Cancer" [24]; and in my books, "The Cancer Microbe" [25], and "Four Women Against Cancer" [26].

Color photographs of Cantwell's cancer bacteria can be viewed on-line at the Journal of Independent Medical Research website ( There are also videos of my lectures on the cancer microbe available on

During the 1970s and 1980s I identified bacteria in acid-fast stained tissue sections from HD patients. Microphotographs of bacteria in tissue sections of the heart, lung, lymph nodes, skin, pancreas, cerebrum, bone and marrow from four HD patients, were reported in 1981, along with a review of the previous literature implicating bacteria in HD [27].

In 1979 I encountered a 56-year-old white man with a very rare HD tumor initially confined to the skin. Figure 2 shows the bacteria deep in the skin, and identified in specially-stained acid-fast tissue sections.

Later that year, new swellings of lymph nodes were also diagnosed as HD. Similar bacteria were observed. Despite radiation therapy and chemotherapy, he died from cardiac arrest the following year. At autopsy, the heart showed changes consistent with "radiation pericarditis."

Rare foci of scattered acid-fast coccoid forms were noted in the lung and heart. The cancer was "cured" in that there was no autopsy evidence of HD. However, microbes were still present in the lung tissue sections, as reported in 1984, by Cantwell and Kelso [28]. More photographs of microbes in HD can be found on the website (

Newer research implicating bacteria in Hodgkin's disease

Could the cancer establishment, except for a few dissidents, be wrong in ignoring and rejecting a bacterial cause of HD?

In this regard, it is important to recall that the Nobel Prize in medicine was awarded in 2005 to two Australian researchers who discovered that stomach ulcers were caused by bacteria that millions of people carry normally in their stomach.

For a century these ulcer-causing bacteria, now identifiable in tissue with a special tissue stain, went undetected by physicians, all of whom were taught that bacteria could not survive in the acid environment of the stomach.

Now a curative antibiotic treatment has been designed to treat Helicobacter pylori infection.

In 1975, using the electron microscope, Parmley et al. showed "microorganism-like structures" in lymph nodes in some untreated patients with HD [29].

More recently, Swiss oncologist Christian Sauter and pathologist Michael Kurrer discovered microbe-like "intercellular rods" and "spheres" in six HD patients [30]. The physicians claim that many features of HD suggest a bacterial infection; and that the epidemiology of HD also suggests a bacterial disease like TB. Sauter and Blum also viewed regression of HD of the lung by use of prolonged antibiotic therapy with ciprofloxacin and clarithromycin [31].

Sauter thinks antibiotic treatment of very early Hodgkin's disease may be successful before there is a genetic exchange between the bacteria and human cells.

Why do cancer experts ignore bacteria in HD and cancer?

Most people do not envision the human body as immersed in a sea of microbes from internal and external sources; and we are told that our only protection from the trillions of potentially dangerous bacteria that inhabit our bodies is our immune system.

Rowan Hooper, writing in Wired News about new research at Imperial College London, notes: "Most of the cells in your body are not your own, nor are they even human. They are bacterial. From the invisible strands of fungi waiting to sprout between our toes, to the kilogram of bacterial matter in our guts, we are best viewed as walking 'superorganisms,' highly complex conglomerations of human cells, bacteria, fungi and viruses. More than 500 different species of bacteria exist in our bodies, making up more than 100 trillion cells. Because our bodies are made of only some several trillion human cells, we are somewhat outnumbered by the aliens. It follows that most of the genes in our bodies are from bacteria, too. Luckily for us, the bacteria are on the whole commensal, sharing our food but doing no real harm." There is also recent evidence that bacteria and human cells constantly "swap genes", much like the AIDS retrovirus swaps its genetic material with human cells.

Despite all that has been published on the cancer microbe, I am aware of microbiologists and pathologists and cancer experts who demand "proof" that these tiny round microscopic forms are indeed bacteria and infectious agents.

But these forms can be seen in the earliest phases of cancer, and when the cancer appears in new areas and, most importantly, can be seen abundantly at autopsy. Nevertheless, most doctors are adamant in their belief that bacteria are not involved in the cause of cancer. (Yet, amazingly, most believe in viruses, even thought most doctors have never seen one.) I contend that after attending medical school physicians should be able to recognize bacteria when they see them. Surely these "forms" reported for a century should be recognized and deserve careful study.

The disinterest of the medical and microbiologic community in investigating bacteria in HD and other forms cancer is not in the tradition of good science.

The microbiology of cancer and the Internet

The bacterial cause of cancer has a rich history dating back to the nineteenth century. Anyone interested in the bacterial cause of cancer can now easily research it on the computer.

An Internet search, using key words such as: cancer microbe, cancer bacteria, pleomorphism, and nanobacteria + cancer, provides a good introduction to the microbiology of cancer.

In addition, I suggest Googling cancer research workers, such as Virginia Livingston, Erik Enby, Guenther Enderlein, Alan Cantwell, Lida Mattman, Wilhelm Reich + T Bacilli, Raymond Royal Rife, and others.

There is no longer any excuse to be ignorant of research pointing to bacteria as a possible cause of cancer, particularly when evidence of such bacteria resides in the medical literature.

Previously, the contents of medical journals were closed to the public because most people were not granted access to , medical libraries. Now all that has changed.

By use of the PubMed website, published medical literature is now easily available to everyone via the click of a mouse.

If the health authorities can conduct a national search for a boy with Hodgkin's cancer and force him to endure radiation and chemotherapy, they can certainly can spend a little time searching for the bacteria that are so clearly visible in Hodgkin's and other forms of cancer.


1. Centkowski P, Sawczuk-Chabin J, Prochorec M, Warzocha K. Hodgkin's lymphoma and tuberculosis coexistence in cervical lymph nodes. Leuk Lymphoma. 2005 Mar;46(3):471-5.

2. Jaffe ES, Zarate-Osorno A, Kingma DW, Raffeld M, Medeiros LJ. The interrelationship between Hodgkin's disease and non-Hodgkin's lymphomas. Ann Oncol. 1994;5 Suppl 1:7-11.

3. Busni N. Uber die Verwantschaft der Mycosis fungoides und der Lymphogranulomatose. Darstellung von Mikroorganismen in Geweben bei experimentellan Granulomen. Virchow's Arch Pathol Anat. 1931; 280:627-639.

4. Busni N. Ein Beitrag zur Atiologie der Lymphogranulomatose. Virchow's Arch Pathol Anat. 1928;268:614-628

5. Aplas V. Tierexperimentelle Untersuchungun zur Virusatiologie der Mycosis fungoides. Archiv Klin Exp Dermatol. 1963; 205:272-281.

6. Aplas V. Weiterer Beitrag sur Atiologie der Mycosis fungoides. Arch Klin Exp Dermatol. 1963; 216:63-70.

7. Cantwell AR. Variably acid-fast pleomorphic bacteria as a possible cause of mycosis fungoides: a report of a necropsied case and two living patients. J Dermatol Surg Oncol. 1982 Mar;8(3):203-

8. Cantwell AR. Variably acid-fast bacteria in a rare case of coexistent malignant lymphoma and cutaneous sarcoid-like granulomas. Int J Dermatol. 1982 Mar;21(2):99-106.

9. Wood C, Harrington W Jr. AIDS and associated malignancies. Cell Res. 2005 Nov-Dec;15(11-12):947-52.

10. Cantwell AR. Kaposi's sarcoma and variably acid-fast bacteria in vivo in two homosexual men. Cutis. 1983 Jul;32(1):58-61, 63-4, 68.

11. Cantwell AR. Necroscopic findings of variably acid-fast bacteria in a fatal case of acquired immunodeficiency syndrome and Kaposi's sarcoma. Growth. 1983 Summer;47(2):129-34.

12. Cantwell AR, Rowe L. African "eosinophilic bodies" in vivo in two American men with Kaposi's sarcoma and AIDS. J Dermatol Surg Oncol. 1985 Apr;11(4):408-12.

13. Cantwell AR. Mycobacterium avium-intracellulare infection and immunoblastic sarcoma in a fatal case of AIDS. Growth. 1986 Spring;50(1):32-40.

14. Aisenberg AC. Problems in Hodgkin's disease management.

Blood. 1999 Feb 1;93(3):761-79.

15. Travis LB, Hill D, Dores GM, Gospodarowicz M, van Leeuwen FE, Holowaty E, Glimelius B, Andersson M, Pukkala E, Lynch CF, Pee D, Smith SA, Van't Veer MB, Joensuu T, Storm H, Stovall M, Boice JD Jr, Gilbert E, Gail MH. Cumulative absolute breast cancer risk for young women treated for Hodgkin lymphoma. J Natl Cancer Inst. 2005 Oct 5;97(19):1428-37.

16. Bowers DC, McNeil DE, Liu Y, Yasui Y, Stovall M, Gurney JG, Hudson MM, Donaldson SS, Packer RJ, Mitby PA, Kasper CE, Robison LL, Oeffinger KC. Stroke as a late treatment effect of Hodgkin's Disease: a report from the Childhood Cancer Survivor Study. J Clin Oncol. 2005 Sep 20;23(27):6508-15.

17. Wuerthele Caspe (Livingston) V, Alexander-Jackson E, Anderson JA, et al. Cultural properties and pathogenicity of certain microorganisms obtained from various proliferative and neoplastic diseases. Amer J Med Sci. 1950; 220;628-646.

18. Alexander-Jackson E. A specific type of microorganism isolated from animal and human cancer: bacteriology of the organism. Growth. 1954 Mar;18(1):37-51.

19. Diller IC. Growth and morphological variability of pleomorphic, intermittently acid-fast organisms isolated from mouse, rat, and human malignant tissues. Growth. 1962; 26:181-209.

20. Diller IC, Diller WF. Intracellular acid-fast organisms isolated from malignant tissues. Trans Amer Micr Soc. 1965; 84:138-148.

Seibert FB, Feldmann FM, Davis RL, Richmond IS: Morphological, biological, and immunological studies on isolates from tumors and leukemic bloods. Ann N Y Acad Sci. 1970 Oct 30;174(2):690-728

21. Seibert FB, Yeomans F, Baker JA, Davis RL, Diller IC. Bacteria in tumors. Trans N Y Acad Sci. 1972 Jun;34(6):504-33.

22. Seibert FB, Farrelly FK, Shepherd CC. DMSO and other combatants against bacteria isolated from leukemia and cancer patients.Ann N Y Acad Sci. 1967 Mar 15;141(1):175-201.

23. Livingston (Wuerthele Caspe) V. Cancer, A New Breakthrough. Los Angeles: Nash Publishing Corp: Los Angeles; 1972.

24. Livingston-Wheeler VWC, Addeo E. The Conquest of Cancer. New York:Franklin Watts; 1984.

25. Cantwell A. The Cancer Microbe. Los Angeles: Aries Rising Press; 1990.

26. Cantwell A. Four Women Against Cancer. Los Angeles: Aries Rising Press; 2005.

27. Cantwell AR. Histologic observations of variably acid-fast coccoid forms suggestive of cell wall deficient bacteria in Hodgkin's disease: a report of four cases. Growth. 1981 Autumn;45(3):168-87.

28. Cantwell AR Jr, Kelso DW. Variably acid-fast bacteria in a fatal case of Hodgkin's disease. Arch Dermatol. 1984 Mar;120(3):401-2.

29. Parmley RT, Spicer SS, Pratt-Thomas HR, Morgan SK, Othersen HB. Microorganism-like structures in Hodgkin disease. Electron microscopical demonstration. Arch Pathol. 1975 May;99(5):259-66.

30. Sauter C, Kurrer MO. Intracellular bacteria in Hodgkin's disease and sclerosing mediastinal B-cell lymphoma: sign of a bacterial etiology? Swiss Med Wkly. 2002 Jun 15;132(23-24):312-5.

31. Sauter C, Blum S. Regression of lung lesions in Hodgkin's disease by antibiotics: case report and hypothesis on the etiology of Hodgkin's disease. Am J Clin Oncol. 2003 Feb;26(1):92-4.


Dr. Cantwell is the author of two books on the microbiology of cancer: The Cancer Microbe and Four Women Against Cancer, both available from and Book Clearing House @ 1-800-431-1579.

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